Oral Presentation The Melbourne Immunotherapy Network Winter Symposium 2019

Is there a role for cancer vaccines in the checkpoint and CAR-T immunotherapy era? (#1)

Riccardo Dolcetti 1
  1. The University of Queensland, Woolloongabba, QLD, Australia

Cancer vaccines may unlock the full potential of immune checkpoint inhibitors and increase the still suboptimal response rates obtainable by these drugs. Nevertheless, the potency and antigen-specificity of available cancer vaccines is still unsatisfactory. To overcome these limitations, we developed an innovative and effective nanoparticle targeting platform to leverage the superior antigen presenting capacity of cross-presenting dendritic cells (DCs), which selectively targets tumour antigens to Clec9A+ cross-presenting DCs in vivo, both in mice and in humans. In a mouse model of HPV-driven carcinoma, Clec9A-targeting Tailored Nanoemulsions (TNEs) loaded with recombinant E6/E7 viral oncoproteins showed a markedly superior efficacy as compared to a standard vaccination protocol. To exploit the tumour “mutanome” with our TNE platform, we developed a functional assay to rank immunogenicity of individual neo-epitopes using the murine B16-F10 melanoma as a model. Four weekly i.v. injections of Clec9A-targeting TNEs loaded with a functionally selected pool of neo-epitopes strongly inhibited the in vivo growth of the highly aggressive and poorly immunogenic B16F10 melanoma with the induction of potent epitope-specific T-cell immunity. The treatment resulted in high rate of tumour eradication and tumour-free mice were protected by a subsequent re-challenge with B16F10 cells, consistent with the induction of strong memory responses.

With the aim to enhance the efficacy of CAR-T cell therapy for solid tumours, we also explored the possibility that Clec9A-TNE could prime DCs for antigen presentation to CAR-T cells through their TCR, thereby enhancing the expansion and activation of adoptively transferred dual-specific T cells. To address this, dual-receptor CAR-T cells, expressing a CAR against the human Her2 antigen, and TCRs specific for Ovalbumin (OVA) peptides SIINFEKL (CAROTI) or OVA323-339 (CAROTII) were combined with an OVA-carrying Clec9A-TNE (OVA-Clec9A-TNE). Combined treatment of mice bearing established Her2+ tumours with CAROT cells and OVA-Clec9A-TNE vaccine led to significant tumour regression and rejection...